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Lipid (cell membrane) damage inhibition of protein synthesis Mitochondria & ATP (cellular energy) depletion hydatidiform mole (molar pregnancy) Fallopian tube issues unexplained infertility miscarriage Pre-eclampsia (high blood pressure during pregnancy) birth defects In addition, and more specifically
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Genetic polymorphisms (SNPs) associated with CFS β’ So far, SNPs in genes for the following proteins have been found to be present at higher frequency in CFS in general or in a subset, either alone or in combination: Immune system: β’ Tumor necrosis factor (TNF) (55) β’ Interferon gamma (IFN-gamma) (55) Neurotransmitter systems: β’ Tryptophan hydroxylase 2 (TPH2) (56,57) β’ Serotonin transporter (5-HTT) gene promoter (58) β’ Serotonin receptor subtype HTR2A (25) β’ Monoamine oxidase A (MAO A) (56) β’ Monoamine oxidase B (MAO B) (56) β’ Catechol-O-methyltransferase (COMT) (57) HPA Axis: β’ Angiotensin converting enzyme (ACE) (59) β’ Proopiomelanocortin (POMC) (56) β’ Corticosteroid binding globulin (CBG) (60) β’ Glucocorticoid receptor--nuclear receptor subfamily3, group C, member 1 (NR3C1) (56, 57, 61) All of these proteins play roles in the pathogenesis described by this hypothesis
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