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The primary pharmacological target of anti-PD drugs is the dopaminergic system (Fig

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Conclusions Some defects in CoQ biosynthesis induce an increase in life span in animal models, most likely due to either a mild reduction in the flow of electrons in the mitochondrial respiratory chain and the subsequent decrease in ROS production or early mitochondrial dysfunction and physiological adaptation to this condition—the latter phenomenon being compatible with the concept of mitohormesis