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In conditions of increased oxidant stress, however, the contribution of the pentose shunt may be significantly increased

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The canonical UPR pathways—inositol-requiring enzyme 1α (IRE1α), protein kinase RNA-like ER kinase (PERK), and activating transcription factor 6 (ATF6)— activate transcription of ER chaperones to help clear misfolded proteins, whether through re-folding or shuttling them for ER-associated degradation (ERAD)