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Deletion of the Tnfaip3/A20 gene in DCs activates the NF-κB pathway, leading to increased numbers of cDC1, cDC2, and monocyte-derived DCs (mo-DCs), as well as upregulated expression of surface activation markers MHC Ⅱ, CD40, and PDL1, thereby inducing PH in mice accompanied by pulmonary vascular remodeling, inflammatory cell infiltration, and release of cytokines such as IL-6

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