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During the pivotal inflammatory cascade activation phase, a sophisticated arsenal of anthraquinone constituents, exemplified by rhein and physcion, implements strategic suppression of NF-κB and STAT3 signaling networks, while hirudin provides targeted inhibition of NLRP3 inflammasome activation, and triterpenoid compounds orchestrate comprehensive modulation of TLR4/MyD88 cascades, collectively establishing a coordinated attenuation of pro-inflammatory mediator expression throughout the entire gut-kidney axis continuum