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Investigations into the neurobiological underpinnings of anxiety-like behaviors in AD models have suggested multiple factors, such as Aβ deposition, Tau pathology, neuroinflammation, and neuronal or neurotransmitter changes

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Type 2 diabetes candidate genes, including PAX5 , were recently validated for functional relevance in human islets, further underscoring the value of experimental follow-up in disease-relevant cell types [14]