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Tumor-associated macrophages can secrete transforming growth factor beta (TGF-β) to turn on hepatic leukemia factor (HLF), which enhances the transcription of gamma-glutamyltransferase 1 (GGT1) and promotes resistance to ferroptosis in TNBC [16]

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Cancer cells co-opt the neuronal redox-sensing channel TRPA1 to promote oxidative-stress tolerance

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